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Chen, J.-A

Indeed, Sp was able to restore cytosolic and mitochondrial redox balance in neuronal cell line SHSY-5Y subjected to 1-methyl-4-phenylpyridinium (MPP + ) treatment which is known to cause mitochondria dysfunction and oxidative stress through complex I inhibition 1 (Fig

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This study uncovers a functional link between altered iSPN BDNF-TrkB signalling, glutathioneascorbate metabolism and hyperdopaminergic state, underscoring the vital role of GSTO2 in maintaining dopamine balance

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